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About felix

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  1. Hello Guys am just keen to have your thoughts on this issue.The trust i work for(Name Witheld) is planning a new build to accommodate all units in one premises and will be ready in 2012.Plans for the New Build do not include any offices for consultants or other professionals. It would be helpful to have your thoughts on those responsibilities and tasks you might not be able to fulfil without your own office. Thanks
  2. People need to be aware that you do not need the MRCPsych to get on to the specialist register these days. Thanks to the emergence of PMETB. Being on the register is what counts nowadays. You can pursue other postgraduate qualifications like MSC Pschiatry, LLM Medical Law and Ethics etc. I am doing the latter. The Irish College of Psychiatrists will soon commence their own membership exams. I obtained my DPM in Ireland and i tell you the Irish are good at assessing your knowledge of basic and clinical sciences and are more keen about your knowledge rather than statistics or paying the mortgage of their headquarters.
  3. I have been sitting the MRCPsch 2 since i passed the part 1 in Nov. 2002. I have come to the conclusion that t is all a farce. It is aPavlovlian experiment. The you do it the more you think that the next examination is your best chance. Like somebody wrote it is not about your performance as an individual, they just set an arbitrary cut off mark which s predetermined and self serving. Do people not think that a CLASS ACTION IIS THE WAY FORWARD even if it is just to force the college to come to the court to explain under oath how the exams are conducted This is on the back of the GMC being taken to the employment tribunal by a medical student over MCQ exams. http://www.dailymail.co.uk/news/article-1039495/Medical-student-dyslexia-takes-court-action-exempted-multiple-choice-tests.html I believe that a class action is the only way to seek redress for an exam that have been reduced to lottery to pay for the mortgage at Belgravia
  4. Morel....Demence precoce Kahlbaum.....Katatonie Hecker....Hebephrenie Kraepelin.....Dementia praecox Bleuler................Schizophrenia splitting of the mind
  5. And of course you have to zero it down to Psychoses and Schizophrenia in particular How does that sound
  6. I think that essay question should address 1. Have adult patents with mental disoerders got significant physical health issues how does it compare with the generl population 2. Are these issues given the attention that they deserve 3. How has the situation arisen 4. What is the current arrangement in addressing physical health and is it satisfactory 5. What needs to change Remember this essay is a repeat See this APT http://apt.rcpsych.org/cgi/content/full/11/2/125 http://apt.rcpsych.org/cgi/content/full/11/6/457 http://apt.rcpsych.org/cgi/content/full/10/2/107
  7. and suicidal
  8. go to www.holiday-inn.co.uk and book a room
  9. check this out http://www.nhslothian.scot.nhs.uk/hospitals/reh.asp http://www.specialistinfo.com/thget.php?t=t_chst&r=LOTPJ2
  10. the same applies to me too. please contact me asap at sept2701@yahoo.co.uk
  11. http://www.rcpsych.ac.uk/exams/regulationsandcurricula/examregulations/examformat/structuredpatientmanagement.aspx http://www.rcpsych.ac.uk/pdf/semOAP_ch21.pdf
  13. Longitudinal COHORT STUDY I SUPPOSE was it not?
  14. 256..True Overall, the age-specific mortality rates for CJD are as expected [3,4] with an increase in incidence up to middle age and highest mortality rates in the 60-to-69 and 70-to-79 years age-groups. The mortality rates in the very elderly are higher than expected in all countries, and it is possible that this reflects improved ascertainment of cases in this population. Another important finding is the high relative incidence rate in the youngest age group (<39 years) in the United Kingdom, which is even more pronounced if the age distribution is analyzed by age at onset of disease. This excess is due to the occurrence of new-variant CJD [16]. Indeed the availability of contemporary comparative data on the incidence of CJD in the younger population was crucial to the hypothesis that these cases might be causally linked to BSE. The collaborative study of CJD in Europe has allowed comparative information on the clinical, pathological, and epidemiological characteristics of CJD to be defined. Although there is no statistical evidence for a causal link between BSE and CJD in this study, the prolonged incubation periods in prion diseases indicate that BSE-related changes in mortality of CJD could still occur. Recent evidence [17, 18] has strengthened the hypothesis that there may be a causal link between BSE and new-variant CJD, and the availability of this baseline information may be of crucial importance in the interpretation of data from continuing epidemiological research in Europe.
  15. 125 T The MRI is generally the most useful supportive diagnostic test in variant CJD. It is a relatively non invasive investigation that is generally readily available and, importantly, is undertaken in cases of suspect variant CJD in order to exclude other possible illnesses. However, there is a characteristic abnormality seen in the posterior thalamic region (the so called “pulvinar sign”) which is highly sensitive and specific for variant CJD. The pulvinar sign has been found in 90+% of pathologically proven vCJD cases. Present indications are that FLAIR sequences are most likely to show the abnormality. The stage of illness at which the scan becomes positive is uncertain. However, individual patients generally have MR scans at a time when clinicians feel they are appropriate and, as indicated above, partly to exclude other illnesses. It is at this clinically indicated point that positive scans have been found to be positive. In a few patients, the pulvinar sign has been present on MRI within 3 months of symptom onset. The finding of a positive MRI scan allows a “possible variant CJD” case to be elevated to the category of “probable variant CJD”. Figure: Transverse FLAIR MRI showing bilateral and symmetrical high signal in the pulvinar nuclei of the thalamus - the 'pulvinar sign' of variant CJD.